Angiogenesis

Angiogenesis

血管生成

  • 1区 中科院分区
  • Q1 JCR分区

期刊简介

《Angiogenesis》是由Springer Netherlands出版社于1997年创办的英文国际期刊(ISSN: 0969-6970,E-ISSN: 1573-7209),该期刊长期致力于外周血管病领域的创新研究,主要研究方向为PERIPHERAL VASCULAR DISEASE。作为SCIE收录期刊(JCR分区 Q1,中科院 1区),本刊采用OA未开放获取模式(OA占比0.4374...%),以发表外周血管病领域等方向的原创性研究为核心(研究类文章占比85.19%%)。凭借严格的同行评审与高效编辑流程,期刊年载文量精选控制在27篇,确保学术质量与前沿性。成果覆盖Web of Science、Scopus等国际权威数据库,为学者提供推动医学领域高水平交流平台。

投稿咨询

投稿提示

Angiogenesis审稿周期约为 较慢,6-12周 。该刊近年未被列入国际预警名单,年发文量约27篇,录用竞争适中,主题需确保紧密契合医学前沿。投稿策略提示:避开学术会议旺季投稿以缩短周期,语言建议专业润色提升可读性。

  • 医学 大类学科
  • English 出版语言
  • 是否预警
  • SCIE 期刊收录
  • 27 发文量

中科院分区

中科院 SCI 期刊分区 2023年12月升级版

Top期刊 综述期刊 大类学科 小类学科
医学
1区
PERIPHERAL VASCULAR DISEASE 外周血管病
2区

中科院 SCI 期刊分区 2022年12月升级版

Top期刊 综述期刊 大类学科 小类学科
医学
1区
PERIPHERAL VASCULAR DISEASE 外周血管病
1区

JCR分区

按JIF指标学科分区 收录子集 分区 排名 百分位
学科:PERIPHERAL VASCULAR DISEASE SCIE Q1 3 / 96

97.4%

按JCI指标学科分区 收录子集 分区 排名 百分位
学科:PERIPHERAL VASCULAR DISEASE SCIE Q1 5 / 96

95.31%

CiteScore

CiteScore SJR SNIP CiteScore 排名
CiteScore:21.9 SJR:3.289 SNIP:1.698
学科类别 分区 排名 百分位
大类:Biochemistry, Genetics and Molecular Biology 小类:Clinical Biochemistry Q1 1 / 117

99%

大类:Biochemistry, Genetics and Molecular Biology 小类:Physiology Q1 6 / 193

97%

大类:Biochemistry, Genetics and Molecular Biology 小类:Cancer Research Q1 14 / 230

94%

期刊发文

  • Targeting SMYD2 inhibits angiogenesis and increases the efficiency of apatinib by suppressing EGFL7 in colorectal cancer

    Author: Zhang, Yi; Zhou, Lei; Xu, Yixin; Zhou, Jingyu; Jiang, Tao; Wang, Jiaqi; Li, Chao; Sun, Xiaoxiong; Song, Hu; Song, Jun

    Journal: ANGIOGENESIS. 2023; Vol. 26, Issue 1, pp. 1-18. DOI: 10.1007/s10456-022-09839-4

  • S100A4-dependent glycolysis promotes lymphatic vessel sprouting in tumor

    Author: Li, Anqi; Zhu, Linyu; Lei, Ningjing; Wan, Jiajia; Duan, Xixi; Liu, Shuangqing; Cheng, Yanru; Wang, Ming; Gu, Zhuoyu; Zhang, Huilei; Bai, Yueyue; Zhang, Li; Wang, Fazhan; Ni, Chen; Qin, Zhihai

    Journal: ANGIOGENESIS. 2023; Vol. 26, Issue 1, pp. 19-36. DOI: 10.1007/s10456-022-09845-6

  • MicroRNA-375 repression of Kruppel-like factor 5 improves angiogenesis in diabetic critical limb ischemia

    Author: McCoy, Michael G.; Jamaiyar, Anurag; Sausen, Grasiele; Cheng, Henry S.; Perez-Cremades, Daniel; Zhuang, Rulin; Chen, Jingshu; Goodney, Philip P.; Creager, Mark A.; Sabatine, Marc S.; Bonaca, Marc P.; Feinberg, Mark W.

    Journal: ANGIOGENESIS. 2023; Vol. 26, Issue 1, pp. 107-127. DOI: 10.1007/s10456-022-09856-3

  • Damage to endothelial barriers and its contribution to long COVID

    Author: Wu, Xiaoming; Xiang, Mengqi; Jing, Haijiao; Wang, Chengyue; Novakovic, Valerie A.; Shi, Jialan

    Journal: ANGIOGENESIS. 2023; Vol. , Issue , pp. -. DOI: 10.1007/s10456-023-09878-5

  • Endothelial hyperactivation of mutant MAP3K3 induces cerebral cavernous malformation enhanced by PIK3CA GOF mutation

    Author: Huo, Ran; Yang, Yingxi; Sun, Yingfan; Zhou, Qiuxia; Zhao, Shaozhi; Mo, Zongchao; Xu, Hongyuan; Wang, Jie; Weng, Jiancong; Jiao, Yuming; Zhang, Junze; He, Qiheng; Wang, Shuo; Zhao, Jizong; Wang, Jiguang; Cao, Yong

    Journal: ANGIOGENESIS. 2023; Vol. 26, Issue 2, pp. 295-312. DOI: 10.1007/s10456-023-09866-9

  • Loss of GLTSCR1 causes congenital heart defects by regulating NPPA transcription

    Author: Han, Fengyan; Yang, Beibei; Chen, Yan; Liu, Lu; Cheng, Xiaoqing; Huang, Jiaqi; Zhou, Ke; Zhang, Dandan; Xu, Enping; Lai, Maode; Lv, Bingjian; Cheng, Hongqiang; Zhang, Honghe

    Journal: ANGIOGENESIS. 2023; Vol. 26, Issue 2, pp. 217-232. DOI: 10.1007/s10456-023-09869-6

  • A ribosomal DNA-hosted microRNA regulates zebrafish embryonic angiogenesis

    Author: Yunwei Shi, Xuchu Duan, Guangmin Xu, Xiaoning Wang, Guanyun Wei, Shikui Dong, Gangcai Xie, Dong Liu

    Journal: ANGIOGENESIS, 2019, Vol., , DOI:10.1007/s10456-019-09663-3

  • Prion protein is essential for diabetic retinopathy-associated neovascularization

    Author: Lingyan Zhu, Jixiong Xu, Ying Liu, Tian Gong, Jianying Liu, Qiong Huang, Shane Fischbach, Wenquan Zou, Xiangwei Xiao

    Journal: ANGIOGENESIS, 2018, Vol., , DOI:10.1007/s10456-018-9619-4